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This is done by increasing type 1 collagen in these tissues which promotes healing & not blocking the nerve ending
Lipid intake pathways are upregulated in CSCs, providing energy essential for survival, which explains why interference with GPX4 pathways seems to render CSCs sensitive to ferroptosis (Recalcati et al., 2019
These important interactions are beyond the scope of this initial investigation, but will be incorporated in future studies
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