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As has been demonstrated in TNF transgenic mice models, synovial fibroblasts and intestine myofibroblasts are activated early by TNF-TNFR1 signaling, produce several matrix degrading enzymes and are sufficient targets to induce TNF-driven inflammatory polyarthritis, Crohn's-like inflammatory bowel disease (IBD) and sacroiliitis [27]
Urinary proteomics identifies distinct immunological profiles of sepsis associated AKI sub-phenotypes
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