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The specific mechanism may be by activating VEGF-A / VEGFR1 -mediated AKT and p38/MAPK signaling pathways to upregulate downstream eNOS expression, which may increase the NO product synthesis thus promote angiogenesis of gastric mucosa, and also interact with ER stress
What the research base contains, and what it doesnt Almost everything published on BPC-157 is preclinical
10.1080/09603123.2023.2210109 159 NarkwicheanA.MaaloufW.BaumgartenM.PolanskiL.Raine-FenningN.CampbellB.et al (2017)
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