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Genet 62 , 10441051 (1998)
Chronic treatment with PARP inhibitors increased NAD + levels and mitochondrial respiration in 10-week-old mice and triggered UPR mt and increased mitochondrial respiration and lifespan in worms 109,257
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Both cardiomyocytes and immune cells rely on glucose metabolism to maintain essential functions, and broad inhibition carries risks of insufficient energy supply, systemic metabolic disturbances, and off-target toxicity
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