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As the prevalence of T2DM increases globally and is recognized as a reemerging risk and challenge to manage TB, more research should investigate the possible benefits of using L-GSH as a potential adjunct therapy alongside current anti-TB treatments or even as a preventative strategy for individuals with T2DM with known dysregulation of GSH metabolism and increased oxidative stress

The complex reciprocal reinforcement between metabolic and immunological pathways makes MASH an immunometabolic disease

[65] This nutrient deficiency/disease correlation, however, has been hypothesized to reveal a risk factor for autism that could potentially be addressed through early intervention in infant nutrition