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Epilepsy can have many physical, psychological and social consequences, and many people do not respond to the current medications, so finding ways to reduce this risk is critical. People living with Type 2 diabetes are known to have a higher risk of developing neurological conditions, including epilepsy, especially as they age

Whereas the levels of 1,2-benzoquinone, 11-dehydro-thromboxane B2, 12-KETE, L-malic acid, N2-succinyl-L-ornithine, ornithine, argininosuccinic acid from the fecal metabolites gradually decreased as CKD progressed, chlorohydroquinone, hydroquinone, L-cystine, fecal 12-keto-tetrahydro-leukotriene B4 (12-keto-tetrahydro-LTB4), and L-cysteine levels gradually increased Integrated network analysis based on metabolomics and metagenomics data revealed that Ruminococcus bromii , fecal hydroquinone, and serum creatinine were the major contributors to this network, therefore play major roles in CKD progression

To better visualize the changes between IC-B and PEC cells in ABMR compared with normal tissue, we assessed the following pathways related to glutathione metabolism, and vitamin A and biosynthetic machinery, based on cells expressing genes related to each significantly altered pathway, alongside average expression ( Supplementary Figures 4C, D )