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found that the inhibition of CD36 palmitoylation induces its mitochondrial membrane translocation, accelerates the conversion of low-fat fatty acids to acyl-coenzyme A through interaction with ACSL1, enhances mitochondrial fatty acid oxidation (FAO) in the liver, reduces ROS accumulation, and ultimately alleviates MAFLD lipid deposition and inflammatory responses in HFD-fed mice (153)

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