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The application of 100 nM CAP elicited inward currents, indicating CAP-responsive DRG neurons, and these responses were abolished by pretreatment with 100 nM GLP-1(736) by 52% (Fig
Administration of the CTSB inhibitor CA-074me prior to pancreatitis induction in a rat model significantly reduced intrapancreatic trypsinogen activation
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doi:10.1002/14651858.CD012614.pub2
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