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2005) found that activating GLP-1 receptors decreased testosterone production in an experimental model, raising concern that GLP-1 agonism could theoretically impair Leydig cell function
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These GLP-1 and dual GIP/GLP-1 receptor agonists work by reducing appetite, slowing digestion, and improving metabolic function
The integration of glucagon-mediated energy expenditure with the appetite-suppressive and insulinotropic effects of GIP and GLP-1 agonism is hypothesized to explain the substantial weight loss and metabolic improvements observed in Phase 2 trials
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