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Thus, as oxidation of Hb occurs, even under normal physiological conditions, the antioxidant defenses have a crucial role in the regeneration of functional Hb and maintenance of low metHb levels [11]
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The glucose-lowering effect of MBH-administered glucagon was mediated via GCGRPKA signalling, depended on functional K ATP channels in the MBH and vanished upon hepatic vagotomy [95]
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