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Bornebusch, A

IGF-1 induces skeletal muscle hypertrophy through activation of satellite cells and enhanced protein synthesis. Adams, G.R., American Journal of Physiology-Endocrinology and Metabolism 2

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Therefore, a possible mechanism proposed by the authors is that the activation of the farnesoid X receptor (FXR) by exendin-4 stimulates the expression of FGF15 in the small intestine and inhibits the expression of cytochrome P450 family 7 subfamily A member 1 (Cyp7a1) in the liver through fibroblast growth factor receptor 4 (FGFR4), thereby reducing fecal bile acid excretion and liver bile acid synthesis [75]

Peripheral Contributions MC3R and Nitric Oxide Beyond the central MC4R mechanism, MC3R activation and possible peripheral melanocortin receptor engagement may contribute to PT-141s effects through nitric oxide-related mechanisms in peripheral sexual tissue complementing rather than duplicating the central mechanism