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Metabolic and vascular contributions to ADRD It is important to recognize that the etiology of ADRD is multifactorial and heterogeneous (Iturria-Medina et al., 2016

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Engineering an improved IgG4 molecule with reduced disulfide bond heterogeneity and increased Fab domain thermal stability

It improves glycemic control, delays gastric emptying, and significantly reduces appetite

Studies in knock-out mice confirmed the biological action of [dA(2)]GLP-1/GcG which is related to its ability to target GIP, GLP-1, and glucagon receptors