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Consequent endothelium damage results in increased permeability of the endothelium due to disruption of cellcell junction (18)

The mechanism is straightforward: eating raises blood glucose, which stimulates insulin release, and insulin is a potent activator of somatostatin neurons in the hypothalamus [3]

This pathway has been studied in several in vitro models and is under investigation as a potential therapeutic target in several cancers, including breast, ovarian, liver, and prostate 15,16,17,23

Dietary modifications, such as adjusting meal timing or composition, may further alleviate discomfort during the initial treatment phase