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By suppressing appetite and reducing cravings, these drugs alter the brain's normal reward responses to food
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The outcome was all-cause mortality
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Hyperglycemia and lipotoxicity drive sustained ROS production through NADPH oxidase (NOX4), mitochondrial respiratory chain complexes I and III, and xanthine oxidase, while concurrently depleting antioxidant defenses such as glutathione (GSH), superoxide dismutase (SOD2), and catalase
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