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At the GLP-1R, it produces the same hypothalamic satiety signaling, gastric emptying delay, and beta-cell potentiation as selective GLP-1 agonists
AOD-9604 may address exactly that problem
Du H, Shao M, Xu S, Yang Q, Xu J, Ke H, et al
This is not merely additive: the MOTS-c may amplify or extend the mitochondrial biogenesis signal beyond what training alone achieves, particularly in older adults whose endogenous MOTS-c response to exercise is blunted [2]
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