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Its oral bioavailability is achieved through the incorporation of the absorption enhancer SNAC (N-[8-(2-hydroxybenzoyl)amino]octanoic acid), which works through two mechanisms: (1) temporarily raising the local pH in the stomach to reduce gastric protease activity

doi: 10.1016/s0092-8674(00)81865-6 106 YeoGSHChaoDHMSiegertAMKoerperichZMEricsonMDSimondsSEet al

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IGF-1 LR3 is engineered with an N-terminal extension and Arg3 substitution that reduces IGFBP-3 binding, extending its effective activity substantially longer in preclinical models