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Due to progressive weight gain (137 kg, BMI 54.9 kg/m 2 , WC 140 cm and WHR 1.0.) despite non-pharmacological interventions with the support of a dietician, kinesiologist and psychologist, treatment with semaglutide was introduced
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BRIN-BD11 cells were used throughout this study for their well characterised responses to multiple stimuli, cell culture based convenience and their established intact response to GLP-1R activation both in terms of cAMP and resulting stimulated insulin secretion 36, 37
This glucose-lowering effect has a low intrinsic risk of hypoglycemia, though the risk increases when combined with insulin or sulfonylureas
These studies suggest that GLP-1 and its receptor play a role in the pathophysiology of congenital hyperinsulinism and that targeting the GLP-1 receptor may be an effective treatment approach for this condition ( ATP hyperinsulinism and decreases glucose infusion rate requirements in infants with K ATP hyperinsulinism ( Role of GLP-1 in Hyperinsulinemic Hypoglycemia Secondary to Gastric Surgery Plasma glucose homeostasis, particularly in the post-prandial state is altered after gastric surgery, including gastrectomy, fundoplication, and bariatric procedures ( via increased GLP-1 release potentiating insulin secretion
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