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The trafficking and fusion of some of these organelles, including secondary and azurophilic granules, is regulated by Munc13-4 through its interaction with Rab27a (ref
[5] It is an inborn error of metabolism that predisposes to red blood cell breakdown
In contrast, periosteal cells with Ad/AAV-induced IGF-1 expression maintained a chondrocyte-like phenotype and produced a hyaline-like matrix with intense intracellular HIF-1 staining in the regenerated tissue area
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