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After starvation for 6 h, the cells were left untreated or stimulated with 0.2 nM human recombinant EGF in the presence or absence of the indicated concentrations of gefitinib or aML5-PEG3 for 5 min
We review the current understanding of BAT pathophysiology, particularly focusing on its role as a metabolic regulator in small rodents and also in humans
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