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Accordingly, Hex treatment also failed to upregulate hippocampal BDNF, stimulate hippocampal plasticity, and improve any of the hippocampus-based memories in 5XFAD mice with either whole-body PPAR knockdown or hippocampus-specific PPAR knockdown, indicating an essential role of hippocampal PPAR in rendering Hex-mediated neuroprotection against AD-associated pathologies

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