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These medications work by mimicking the incretin hormone GLP-1, enhancing glucose-dependent insulin secretion, suppressing glucagon release, slowing gastric emptying, and reducing appetite through central nervous system pathways
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GIP Signaling, Insulin Response, and Metabolic Effects GIP receptor activation is part of incretin physiology and can contribute to insulin secretion after meals, which is relevant to type 2 diabetes research [1], [10]
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