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Feb 15 2004;103(4):1364-1369
The catalytic cysteine is highly susceptible to oxidation and nitrosylation, leading to reversible or irreversible modifications that abolish its nucleophilic function and inactivate its enzyme activity (87, 88)
Research Mechanism Mimics hGHs lipolytic domain, activating 3-adrenergic receptors and AMPK pathways to promote lipid oxidation and inhibit lipogenesis
and the National Natural Science Foundation of China (Grant No
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