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Key findings include its ability to promote tendon outgrowth and muscle regeneration, likely through activation of the FAKpaxillin signaling pathway, which is essential for cell migration and adhesion [3]
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Additional triggers for eNOS phosphorylation include vascular endothelial growth factor (VEGF)-induced signal transduction, insulin-mediated activation of PKB/AKT, and bradykinin-induced activation of Ca 2+ /calmodulin-dependent protein kinase II (CaMKII)
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