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Here we introduce a model of synaptic plasticity based on data-constrained postsynaptic calcium dynamics, and show in a neocortical microcircuit model that a single parameter set is sufficient to unify the available experimental findings on long-term potentiation (LTP) and long-term depression (LTD) of PC connections

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Their multiple mechanisms of action, which include significant weight loss, improved glycaemic control, blood pressure reduction, anti-inflammatory effects and potential direct cardioprotective and vascular effects, target multiple pathophysiological pathways implicated in HF progression

Avoid exposure to temperatures above recommended ranges to maintain peptide integrity