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Water, Centella Asiatica Extract (12.3%), Glycerin, Cetyl Ethylhexanoate, Cetearyl Alcohol, Glyceryl Stearate, Glutathione (1,000ppm), Niacinamide (1,000ppm), Madecassic Acid, Asiaticoside, Asiatic Acid, Solanum Melongena (Eggplant) Fruit Extract, Melaleuca Alternifolia (Tea Tree) Leaf Extract, Hydrolyzed Collagen, Beta-Glucan, Hippophae Rhamnoides Fruit Extract, Nelumbo Nucifera Extract, Artemisia Annua Extract, Oryza Sativa (Rice) Extract, Saccharomyces Ferment, Squalane, Sodium Hyaluronate, Dipropylene Glycol, Butylene Glycol, Hydrogenated Lecithin, Cholesterol, Xanthan Gum, Potassium Hydroxide, Disodium EDTA, Caprylic/Capric Triglyceride, Cyclopentasiloxane, Cyclohexasiloxane, Saccharide Hydrolysate, Sorbitan Stearate, Dimethicone, PEG-100 Stearate, Stearic Acid, 1,2-Hexanediol, Chlorphenesin, Carbomer, Caprylyl Glycol *For the most recent ingredient list, please consult the product packaging or contact us for a product photo.
Animal Studies: Strong Results in Obese Rodent Models The foundational animal work was conducted in obese Zucker rats and ob/ob mice, two standard models for studying obesity
It works if you work it
2.1.2 Dysregulation of key iron-regulating proteins In ferroptosis, intracellular iron homeostasis is maintained by a network of regulatory mechanisms, including iron acquisition via the transferrin (Tf)/transferrin receptor 1 (TfR1) pathway, intracellular transport by divalent metal transporter 1 (DMT1), export by ferroportin (FPN1), storage in ferritin, and iron release through ferritinophagy mediated by nuclear receptor coactivator 4 (NCOA4) (39, 40)
Polymorphic light eruption and actinic keratosis: MT1 has been studied in Phase 3 trials for polymorphic light eruption and Phase 2 trials for actinic keratosis and squamous cell carcinoma, reflecting interest in its broader photoprotective potential
