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This review has delineated the anatomical and pathophysiological basis of rotator cuff-associated muscle atrophy, with a particular focus on the pivotal role of ROS in mediating oxidative damage, mitochondrial dysfunction, myofiber apoptosis, and activation of key catabolic signaling cascades, including the FOXO, NF-B, and MAPK pathways
At 300 mcg, which is the standard current protocol, the effect size is likely more modest still
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