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Research has revealed that TGF- stimulates the canonical Wnt signaling pathway in a p38-dependent manner by downregulating the expression of the Wnt antagonist Dickkopf-1 ( Figure 3 details the molecular cascade of the canonical Wnt pathway, highlighting how its activation by TGF- leads to -catenin stabilization and subsequent transcription of pro-fibrotic genes, underscoring its significance as a potential therapeutic target