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Yet, they often have identifiable causes and risk factors
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Finally, a number of inherited mutations in genes involved in carnitine shuttling and fatty acid oxidation pathways do not systematically result in carnitine depletion (such that carnitine supplementation may not help mitigate the symptoms) but lead to abnormal profiles of acylcarnitine esters in blood (35, 40)
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We have earlier, in several steps, developed mathematical models for insulin signalling in human adipocytes: first in isolation and later connected to models of systemic glucose control, and used the models to unravel key alterations in type 2 diabetes [58]
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