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1 When released, GLP-1 activates its receptor, GLP-1R, in the pancreatic islet cells, provoking a myriad of metabolic changes characterized by insulin secretion and appetite suppression

Advances in GLP-1 treatment: focus on oral GLP1-S

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Hansen MK et al

These changes were accompanied by preservation of structural proteins including aggrecan and collagen II, along with reduced caspase-3mediated apoptosis, suggesting a coordinated reduction in glycation-driven cellular stress and tissue degradation

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