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When cells undergo apoptosis-stimulating stress, HtrA2 can either expose inhibitors of the apoptosis protein (IAP) binding motif to mediate cell death or mediate cell death through its serine protease activity [60]

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This limited ability of fiber cells to repair themselves makes the lens susceptible to damage that can lead to the accumulation of protein, lipid, and DNA modifications in nucleated lens cells [67]

When our storage of glutathione drops below optimal levels, we may be at a higher risk of experiencing health problems

Kohr G, Eckardt S, Luddens H, Monyer H, Seeburg PH (1994)