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DNAM-1 has been reported to be an activating receptor on NK cells [152]

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Addressing the mechanism of the H 2 O 2 -induced macrophage death, immunoblotting for cleaved caspase-3, measurement of released IL-1 and IL-18, and use of nercostatin-1 did not support caspase-3-dependent apoptosis, inflammasome activation or necroptosis as the underlying cause of the cytotoxicity, respectively

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