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In models of nerve injury and neuropathic pain, it appears to promote small nerve fiber regeneration and reduce hyperexcitability, which translates into less burning, tingling, and allodynia

The decreased expression of ATGL inhibits fatty acid mobilization of intracellular lipid droplets and promotes lipid droplet's aggregation, leading to renal tubular epithelial cell injury and renal fibrosis, and ultimately promotes the transformation of AKI to CKD[27]

Studies show GHK-Cu binding preferentially to specific DNA sequences suggesting targeted gene regulation rather than random effects

2(1):5861 The potential of GHK as an anti-aging peptide Mortazavi SM, Mohammadi Vadoud SA, Moghimi HR., BioImpacts , 2024

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