From an immunological perspective, these associations arise from a breakdown in self-tolerance mediated by overlapping T-cell and B-cell autoreactivity, shared cytokine pathways (e.g., IFN-, IL-18), and common antigen-presenting HLA alleles
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1.1 Emerging role of dietary interventions Recent research suggests that certain dietary patterns can influence essential pathophysiological characteristics of AD, such as neuroinflammation, oxidative stress, and amyloid-beta deposition (Stefaniak et al., 2022)
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