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Molecular physiology of amylin
D.ZhaoY
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These results indicate that the increased amount of non-12-OH FXR-antagonistic BAs consequent to the decrease in CYP8B1 expression after T3 treatment potentiates GLP-1 production by deactivating the intestinal FXR, thereby improving glucose homeostasis via the augmentation of insulin secretion
To view a copy of this license, visit About this article Cite this article Cheteh, E., Augsten, M., Rundqvist, H
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