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doi: 10.1093/cid/civ085 544 DaminMRCortes-PerezNGQuintanaETOrtiz-MorenoAGarfias NoguezCCruceo-CasarrubiasCEet al
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Defective HR engendered by the PDS5B 17A mutation was also reflected in the sensitivity of mutant cells to the PARP inhibitor Rucaparib, though this sensitivity was not as severe as that observed with BRCA2 knockdown (Supplementary Fig
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