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However, the Shh pathway is activated in response to renal injury, particularly in damaged TECs [104]

on the other hand, the restored autophagy system accelerates the clearance of damaged mitochondria (mitophagy), blocks the continuous generation of ROS, thereby downregulating the Bax/Bcl-2 ratio and inhibiting Caspase-3/7 activity, ultimately significantly reducing the apoptosis level of renal tubular epithelial cells

The other additional proteins including O-acetylserine sulfhydrylase-A, O-acetylserine sulfhydrylase-B, and MalY were also identified to have l-cysteine desulfhydrase activity [22]

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