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One mechanism of dioxin-induced liver toxicity is the activation of the AhR in the hepatocytes, which results in a continuous production of higher levels of ROS and a significant increase in lipid droplets in the liver, producing a similar change observed in NAFLD (Furue et al

It is a structural analogue of alpha-melanocyte-stimulating hormone (alpha-MSH)

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The pQCT measurements in vitro revealed that the increase in the cortical BMC was due to an increased cross-sectional bone area, whereas the cortical volumetric BMD was unchanged