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Furthermore, these treatments mostly target the disease's advanced stages and fall short in addressing the underlying inflammation and oxidative stress [26]

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It operates through nutritional biochemistry rather than receptor signaling

Doi: Garca-Cerro S, Rueda N, Vidal V, Lantigua S, Martnez-Cu C, Garca-Cerro S, Rueda N, Vidal V, Lantigua S, Martnez-Cu C (2017) Normalizing the Gene Dosage of Dyrk1A in a Mouse Model of Down Syndrome Rescues Several Alzheimers Disease Phenotypes