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KPV exerts its anti-inflammatory effects through intracellular pathways, independent of melanocortin receptors (MC-Rs)

This occurs because the condition disrupts the countercurrent heat exchange mechanism between the pampiniform plexus and the central arterial system, resulting in reversed venous flow (55)

This cellular defense mechanism against ferroptosis operates independently of GPX4 and FSP1 (Fig

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Uncontrolled misuse can lead to cardiovascular events.Hormonal Effects: Side effects can include increased sexual desire, acne, increased hair growth, and voice changes