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90 KamimotoM.MizunoS.NakamuraT
Homocysteine reduces nitric oxide availability in endothelial cells Homocysteine impairs the transport of L-arginine, the precursor of NO into endothelial cells and increases the production of ROS by NADPH oxidase, which may cause endothelial nitric oxide synthase (e-NOS) uncoupling
Therefore, N-acetylcysteine (NAC) is an effective antidote for APAP poisoning because it provides extra cysteines for GSH synthesis in the liver
In vivo, cLip-1 increased the survival of mice in which acute renal failure was induced through genetic deletion of Gpx4 , which encodes the ferroptosis suppressor glutathione peroxidase 4 (GPX4) 2 (Fig
Characterization of a Drosophila glutathione transferase involved in isothiocyanate detoxification
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