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Description

These findings suggest that glutamate/GABA imbalance, particularly within the corticostriatal pathway, may serve as a key pathophysiological mechanism and potential biomarker in SHANK3-associated ASD

Docosahexaenoic (DHA) modulates phospholipid-hydroperoxide glutathione peroxidase (Gpx4) gene expression to ensure self-protection from oxidative damage in hippocampal cells