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Recently, a significant role for GSTO1 in protein deglutathionylation has been described 11 , and -actin was identified as one of the cellular substrates for GSTO1-catalysed deglutathionylation
Mutations in SLC6A1 result in partial or complete loss of GABA uptake function, causing ASD
As a result, these effects reduce the integrity of yeast cell membranes, thereby accelerating cell death
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Activation of high-affinity uptake of glutamate by phorbol esters in primary glial cell cultures
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