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In genetic obesity mouse models, systemic suppression of BCAA catabolic gene expression can be observed, with elevated BCKDH phosphorylation (inactivation) levels, and these metabolic abnormalities often precede overt insulin resistance, suggesting that BCAA metabolic dysfunction may represent an early event in obesity-related metabolic disorders
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2,4-Dienoyl-CoA reductase regulates lipid homeostasis in treatment-resistant prostate cancer
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